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Uterine Fibroids 14-Day Meal Plan & Recipes

Uterine Fibroids 14-Day Meal Plan & Recipes

Effects of 6 Months of Soy-Enriched High Protein Compared to Eucaloric Low Protein Snack Replacement on Appetite, Dietary Intake, and Body Composition in Normal-Weight Obese Women: A Randomized Controlled Trial

Lutein Has a Positive Impact on Brain Health in Healthy Older Adults: A Systematic Review of Randomized Controlled Trials and Cohort Studies

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A Day In The Life Of The Ultimate Fertility Diet

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By Sadia Afrin 1, † , Abdelrahman AlAshqar 1, 2, † , Malak El Sabeh 1 , Mariko Miyashita-Ishiwata 1 , Lauren Reschke 1 , Joshua T. Brennan 1 , Amanda Fader 1 and Mostafa A. Borahay 1, *

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Multiple Fibroids Removed From The Uterus Of A 39 Year Old Woman In Mumbai

A healthy lifestyle and a balanced diet play a paramount role in promoting and maintaining homeostatic functions and preventing an array of chronic and debilitating diseases. Based upon observational and epidemiological investigations, it is clear that nutritional factors and dietary habits play a significant role in gynecological disease development, including uterine leiomyoma, endometriosis, polycystic ovary syndrome, and gynecological malignancies. Diets rich in fruits and vegetables, Mediterranean diets, green tea, vitamin D, and plant-derived natural compounds may have a long-term positive impact on gynecological diseases, while fats, red meat, alcohol, and coffee may contribute to their development. Data regarding the association between dietary habits and gynecological disorders are, at times, conflicting, with potential confounding factors, including food pollutants, reduced physical activity, ethnic background, and environmental factors limiting overall conclusions. This review provides a synopsis of the current clinical data and biological basis of the association between available dietary and nutritional data, along with their impact on the biology and pathophysiology of different gynecological disorders, as well as an outlook on future directions that will guide further investigational research.

Worldwide, women are afflicted by a spectrum of gynecological disorders, ranging from benign entities, such as uterine leiomyoma, endometriosis, and polycystic ovary syndrome, to various gynecological malignancies. These disorders represent a significant source of morbidity by causing bothersome heavy menstruation, debilitating pelvic pain, chronic anovulation, hyperandrogenism, infertility, and even death. Although these conditions are discrepant in nature, they share a common feature: they lack a curative medical treatment that would allow the preservation of functioning reproductive organs. Most medications are only temporarily effective, have undesirable side effects, interfere with pregnancy, and carry a risk of disease recurrence upon discontinuation [1, 2, 3]. This stresses the importance of fully understanding the pathophysiology of these disorders and the potential risk factors associated with their development and maintenance to mitigate and modify their consequences on health whenever possible.

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Diet and health have been among the most complex topics in public discourse and scientific circles. There have long been debates regarding the role that nutritional components and dietary habits play in modulating the risk of gynecological diseases, such as uterine leiomyoma [4, 5, 6], endometriosis [7, 8], polycystic ovary syndrome (PCOS) [9, 10], and different gynecological malignancies [11, 12, 13, 14], with the majority of evidence retrieved from epidemiological studies. Evidence suggests that a diet rich in fruit and vegetables, green tea, vitamins, and plant-derived compounds may help prevent gynecological disorders, compared to a diet deficient in vegetables and fruit and high in animal or dietary fats, red meat, and alcohol [6, 7, 12, 14]. Understanding the role of diet in gynecology will change our perspective of how common gynecological diseases develop, progress, and lead to substantial adverse effects on women and will guide us toward pioneering novel diagnostic and therapeutic frameworks to reduce their burden. Most importantly, implementing preventive strategies aimed at changing certain dietary habits may ameliorate the occurrence of a wide array of gynecological diseases. Herein, we reviewed the latest research regarding the role of diet and nutrition in gynecological disorders, with an emphasis on clinical and epidemiological studies.

What Causes Uterine Fibroids?

This article provides a comprehensive review of the available literature discussing the role of diet and nutrition in the biological development of various gynecological disorders, emphasizing clinical and epidemiological data. A literature search was conducted using electronic databases, including PubMed of the National Library of Medicine, Google Scholar, Web of Science, and relevant clinical trials addressing diet and nutrition and gynecological disorders. The keywords “diet”, “nutrition”, “fruit”, “vegetables”, “vitamin”, “fat”, “meat”, “fish”, “alcohol”, “coffee”, “tea”, “grain”, “fiber”, “dairy” and “natural compound” combined with “uterine leiomyoma”, “uterine fibroid”, “endometriosis”, “polycystic ovary syndrome”, and “gynecological malignancy” were used. All relevant reports were retrieved, and the corresponding reference lists were systematically searched to identify any additional studies that could be included. Only papers published as full-length articles in English were considered.

The

Dietary patterns differ in the type of nutritional constituent they contain. Whole grains, such as brown rice, barley, and wheat, are rich in minerals, vitamins, fibers, and phytochemicals, including vitamin E, carotenoids, inulin, and lignans, which play notable roles in modulating immune responses and oxidative stress [15]. Nuts, on the other hand, are rich in healthy (unsaturated) fats, which have been shown to confer a lower risk of obesity, diabetes, and cardiovascular disease. Fruit and vegetables are a known source of vitamins, minerals, and dietary fibers, many of which are notable antioxidants, such as β-carotene, vitamin C, and vitamin E; meat is high in protein and saturated (unhealthy) fatty acids and is a source of N-nitroso compounds, heterocyclic amines, and polycyclic aromatic hydrocarbons, which are known mutagens. Fish, by contrast, provides dietary protein, minerals, and omega-3 fatty acids, which are implicated in reducing the risk of cardiovascular disease [15]. Green tea has been extensively studied for its beneficial effects, and its component epigallocatechin-3-gallate (EGCG), a catechin, has received a great deal of attention for its antioxidant and anti-tumorigenic properties [16]. Later in this review, we will elaborate in depth how different food components and dietary constituents influence the occurrence of common disorders in gynecology.

Our daily diets consist of a myriad of nutritional constituents that have indispensable roles in maintaining physiological processes by participating in intricate homeostatic cascades. It has been well established that deficiencies in certain nutrients and excess of others can have detrimental consequences by means of disrupting physiology and promoting disease. Among all, micronutrients, consisting of minerals, vitamins, and other trace elements, seem to possess particularly vital roles on cellular grounds. For example, zinc and selenium participate as cofactors in modulating enzymatic activity in hundreds of pathways, whereas zinc has an additional role in gene transcription, depicting their profound effects in regulating cellular functions [17]. Vitamins play an essential role in maintaining health and preventing disease. Tocopherols (vitamin E) and carotenoids (vitamin A) are remarkable in their potential to scavenge reactive oxygen species (ROS), which alongside other zinc- and selenium-containing enzymes, such as glutathione peroxidase and superoxide dismutase, are potent antioxidants. Furthermore, riboflavin and niacin participate in the electron transport chain and cellular energy production, whereas folic acid is involved in DNA synthesis and its deficiency has long been implicated in fetal neural tubal defects in pregnancy [18]. The actions of micronutrients extend to affect the immune system. With regard to the innate immune barrier, iron and zinc are responsible for sustaining epithelial integrity and repair, while calcitriol (vitamin D) promotes a healthier composition of the intestinal microbiota in the gut barrier. On the other hand, vitamins C, E, and B6 enhance T cell differentiation and proliferation and antibody production, emphasizing the prominent role of diet in combating infections [19].

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The Uterine Fibroid Diet

Moving to macronutrients, dietary fats have been shown to have both beneficial and adverse effects on human physiology. On the one hand, trans-fatty acids accentuate inflammation and oxidative stress and influence autophagy and apoptosis. For example, it has been shown that trans-fatty acids potentiate the levels of inflammatory mediators, such as C-reactive protein (CRP) and interleukin (IL) 6, predisposing to atherosclerosis [20]. By contrast, omega-3 fatty acids dampen leukocyte chemotaxis and production of various proinflammatory cytokines, suggesting their use as possible therapeutic agents given their anti-inflammatory properties [21]. It has been found that high glucose levels can initiate several pathophysiologic cascades culminating in drastic outcomes, as already seen in patients with diabetes mellitus. While hyperglycemia mediates vasculopathy, neuropathy, and immunosuppression, it is also implicated in tumorigenesis as will be further discussed

This article provides a comprehensive review of the available literature discussing the role of diet and nutrition in the biological development of various gynecological disorders, emphasizing clinical and epidemiological data. A literature search was conducted using electronic databases, including PubMed of the National Library of Medicine, Google Scholar, Web of Science, and relevant clinical trials addressing diet and nutrition and gynecological disorders. The keywords “diet”, “nutrition”, “fruit”, “vegetables”, “vitamin”, “fat”, “meat”, “fish”, “alcohol”, “coffee”, “tea”, “grain”, “fiber”, “dairy” and “natural compound” combined with “uterine leiomyoma”, “uterine fibroid”, “endometriosis”, “polycystic ovary syndrome”, and “gynecological malignancy” were used. All relevant reports were retrieved, and the corresponding reference lists were systematically searched to identify any additional studies that could be included. Only papers published as full-length articles in English were considered.

The

Dietary patterns differ in the type of nutritional constituent they contain. Whole grains, such as brown rice, barley, and wheat, are rich in minerals, vitamins, fibers, and phytochemicals, including vitamin E, carotenoids, inulin, and lignans, which play notable roles in modulating immune responses and oxidative stress [15]. Nuts, on the other hand, are rich in healthy (unsaturated) fats, which have been shown to confer a lower risk of obesity, diabetes, and cardiovascular disease. Fruit and vegetables are a known source of vitamins, minerals, and dietary fibers, many of which are notable antioxidants, such as β-carotene, vitamin C, and vitamin E; meat is high in protein and saturated (unhealthy) fatty acids and is a source of N-nitroso compounds, heterocyclic amines, and polycyclic aromatic hydrocarbons, which are known mutagens. Fish, by contrast, provides dietary protein, minerals, and omega-3 fatty acids, which are implicated in reducing the risk of cardiovascular disease [15]. Green tea has been extensively studied for its beneficial effects, and its component epigallocatechin-3-gallate (EGCG), a catechin, has received a great deal of attention for its antioxidant and anti-tumorigenic properties [16]. Later in this review, we will elaborate in depth how different food components and dietary constituents influence the occurrence of common disorders in gynecology.

Our daily diets consist of a myriad of nutritional constituents that have indispensable roles in maintaining physiological processes by participating in intricate homeostatic cascades. It has been well established that deficiencies in certain nutrients and excess of others can have detrimental consequences by means of disrupting physiology and promoting disease. Among all, micronutrients, consisting of minerals, vitamins, and other trace elements, seem to possess particularly vital roles on cellular grounds. For example, zinc and selenium participate as cofactors in modulating enzymatic activity in hundreds of pathways, whereas zinc has an additional role in gene transcription, depicting their profound effects in regulating cellular functions [17]. Vitamins play an essential role in maintaining health and preventing disease. Tocopherols (vitamin E) and carotenoids (vitamin A) are remarkable in their potential to scavenge reactive oxygen species (ROS), which alongside other zinc- and selenium-containing enzymes, such as glutathione peroxidase and superoxide dismutase, are potent antioxidants. Furthermore, riboflavin and niacin participate in the electron transport chain and cellular energy production, whereas folic acid is involved in DNA synthesis and its deficiency has long been implicated in fetal neural tubal defects in pregnancy [18]. The actions of micronutrients extend to affect the immune system. With regard to the innate immune barrier, iron and zinc are responsible for sustaining epithelial integrity and repair, while calcitriol (vitamin D) promotes a healthier composition of the intestinal microbiota in the gut barrier. On the other hand, vitamins C, E, and B6 enhance T cell differentiation and proliferation and antibody production, emphasizing the prominent role of diet in combating infections [19].

-

The Uterine Fibroid Diet

Moving to macronutrients, dietary fats have been shown to have both beneficial and adverse effects on human physiology. On the one hand, trans-fatty acids accentuate inflammation and oxidative stress and influence autophagy and apoptosis. For example, it has been shown that trans-fatty acids potentiate the levels of inflammatory mediators, such as C-reactive protein (CRP) and interleukin (IL) 6, predisposing to atherosclerosis [20]. By contrast, omega-3 fatty acids dampen leukocyte chemotaxis and production of various proinflammatory cytokines, suggesting their use as possible therapeutic agents given their anti-inflammatory properties [21]. It has been found that high glucose levels can initiate several pathophysiologic cascades culminating in drastic outcomes, as already seen in patients with diabetes mellitus. While hyperglycemia mediates vasculopathy, neuropathy, and immunosuppression, it is also implicated in tumorigenesis as will be further discussed

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